The mechanisms of delayed onset type adverse reactions to oseltamivir
نویسندگان
چکیده
منابع مشابه
The mechanisms of delayed onset type adverse reactions to oseltamivir
Oseltamivir is recommended for the treatment and prophylaxis of influenza in persons at higher risk for influenza complications such as individuals with diabetes, neuropsychiatric illnesses, and respiratory, cardiac, renal, hepatic or haematological diseases. However, a recent Cochrane review reported that reduction of antibody production, renal disorders, hyperglycaemia, psychiatric disorders,...
متن کاملThe mechanisms of sudden‐onset type adverse reactions to oseltamivir
Oseltamivir is contraindicated for people aged 10-19 in principle in Japan, due to concern about abnormal behaviours. Sudden death is another concern. This review examines growing evidence of their association and discusses underlying mechanisms of these sudden-onset type reactions to oseltamivir. First, the importance of animal models and the concept of human equivalent dose (HED) is summarize...
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The rate of infusion reactions to total-dose intravenous iron polymaltose is very low, but the frequency and severity of adverse reactions following the infusion are unknown. In 50 consecutive patients, adverse reactions developed up to 2 days after the infusion in 26% and lasted 1-8 days (median 4). Severe systemic reactions occurred in 8%. Patients should be warned of the chance of delayed re...
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چکیده ندارد.
Fatal neuropsychiatric adverse reactions to oseltamivir: Case series and overview of causal relationships
Background: Infection-associated encephalopathies such as Reye’s syndrome have been one of the major public health problems in many countries. The not dissimilar neuropsychiatric adverse reactions, including deaths, observed with Tamiflu (oseltamivir phosphate: OP) have been another current problem especially in Japan. Methods: Among the cases with neuropsychiatric adverse reactions to Tamiflu ...
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ژورنال
عنوان ژورنال: Infectious Diseases
سال: 2016
ISSN: 2374-4235,2374-4243
DOI: 10.1080/23744235.2016.1189592